@IM_Crit_

@IM_Crit_

imcrit.bsky.social

Intensivist I Internal Medicine | ☕️, 🍩, 🥐, 🍫 addict | #emimcc

👤 Joined on 08 November 2024

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  • 08 Kas 2024, 02:19
    🎉 Bluesky'e katıldı - İlk kullanıcı adı: @imcrit.bsky.social

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Definitely agree. Every trial on diuretic strategies in heart failures show increased creatinine. I don't think we should care about that.

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I think most septic patients do fine without an echo, but "septic" patients do not, and without an echo it's hard to know who is who.

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All vasopressin and dobutamine trials should look at EF, CI/CO and heart rate. No use in adding dobutamine to an already overstimulated heart, likely detrimental to add vasopressin to any heart that isn’t hyperdynamic.

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We do have evidence that response to vasopressin varies depending on EF, and that makes perfect sense. All these trials on differing hemodynamic supportive therapies that do not consider the hemodynamic phenotype are entirely wasted.

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I treat flowtrack like I treat a lactic acid... Sometimes useful, oftentimes too limited by confounding to be helpful. In my practice area I see similar rates of people throwing both these tests/monitors at patients without coming to the bedside to observe the physiology themselves.

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