Cláudio Gomes Lab - Protein Misfolding in Disease
claudiogomeslab.bsky.social
Studying proteins and molecular mechanisms of misfolding and aggregation in Alzheimer's Disease and protein folding disorders at ULisboa | Tau | Amyloid | Chaperones | Proteostasis | Structural Biochemistry | https://folding.campus.ciencias.ulisboa.pt
👤 Joined on 10 March 2025
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- 10 Mar 2025, 21:00
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Amyloid-beta precursor protein (APP) is a complicated protein with many functions. A corrupted form can destroy nerve cells, leading to the loss of thought and memory in Alzheimer's disease. #WorldAlzheimersDay
Show PostA strand of RNA has been discovered to perform two key reactions required for self-replication. The molecule offers a clue towards understanding the origin of life, namely how non-living chemistry on prebiotic Earth started to self-replicate and evolve.
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According to a new #ScienceAdvances study, wheat fiber-enriched foods may reduce gut inflammation and curb the development of inflammatory bowel disease. https://scim.ag/4uU8z1Q
Show PostThe production and accumulation of aberrant proteins can have serious pathological consequences. In a new #ScienceReview, researchers explore how organisms maintain the extracellular proteome. https://scim.ag/4tdEB7B
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Check out this Review published in @science.org whilst I was on vacation Systems-level organization of extracellular proteostasis | Science www.science.org/doi/10.1126/...
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